JNNP

HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS REGISTER
[Advanced]

This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this link to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Add article to my folders
Right arrow Download to citation manager
Right arrowRequest Permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Dietz, V
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Dietz, V
Related Collections
Right arrow Neuromuscular disease
Right arrow Other evidence based practice
Journal of Neurology Neurosurgery and Psychiatry 2003;74:820-821
© 2003 BMJ Publishing Group


LETTER

Spastic movement disorder: what is the impact of research on clinical practice?

V Dietz

ParaCare, Institute for Rehabilitation and Research, University Hospital Balgrist, Forchstr 340, 8008 Zurich, Switzerland

Correspondence to:
Correspondence to:
Professor Dr V Dietz;
dietz@balgrist.unizh.ch

Keywords: spastic paresis; clinical practice

The first 150 words of the full text of this article appear below.

One expects that convincing research results would have an impact on clinical practice. However, whether or not a new concept becomes transferred to an application in clinical practice is dependent on the medical field and on the therapeutic consequences. The issue discussed here concerns spasticity, a common motor disorder in, for example, patients who have had a stroke or a spinal cord injury.


The traditional concept
Over many years it was widely accepted that spasticity consists of muscle hypertonia (that is, "a velocity dependent resistance of a muscle to stretch"1) caused by exaggerated reflexes, leading to the spastic movement disorder.2 This concept was based on animal experiments (for example, in the decerebrate cat3) and on the physical signs evident on clinical examination at the bedside. Consequently, the aim of any treatment was to reduce reflex activity by antispastic drugs. Possible differences in pathophysiology between the clinical signs of spasticity and the . . . [Full text of this article]




This article has been cited by other articles:


Home page
Ann Clin BiochemHome page
G. O'Malley, C. Moran, M. S. Draman, T. King, D. Smith, C. J Thompson, and A. Agha
Central pontine myelinolysis complicating treatment of the hyperglycaemic hyperosmolar state
Ann Clin Biochem, July 1, 2008; 45(4): 440 - 443.
[Abstract] [Full Text] [PDF]


Home page
JRSMHome page
E Ernst
Adverse effects of spinal manipulation: a systematic review
J R Soc Med, July 1, 2007; 100(7): 330 - 338.
[Abstract] [Full Text] [PDF]


Home page
J. Neurol. Neurosurg. PsychiatryHome page
A J Thompson, L Jarrett, L Lockley, J Marsden, and V L Stevenson
Clinical management of spasticity
J. Neurol. Neurosurg. Psychiatry, April 1, 2005; 76(4): 459 - 463.
[Full Text] [PDF]


Home page
NeurologyHome page
E. Montane, A. Vallano, and J. R. Laporte
Oral antispastic drugs in nonprogressive neurologic diseases: A systematic review
Neurology, October 26, 2004; 63(8): 1357 - 1363.
[Abstract] [Full Text] [PDF]


Home page
J. Neurol. Neurosurg. PsychiatryHome page
A A Leis, J A Van Gerpen, J J Sejvar, M Park, R E Bartt, and J S Hui
The aetiology of flaccid paralysis in West Nile virus infection * Authors' reply
J. Neurol. Neurosurg. Psychiatry, June 1, 2004; 75(6): 940 - 941.
[Full Text] [PDF]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS REGISTER
Terms and conditions relating to subscriptions purchased online  ¦  Website terms and conditions  ¦  Privacy policy
Copyright © 2003 by the BMJ Publishing Group Ltd.