|
|
||||||||||||||
|
|
|||||||||||||||
CORRESPONDENCE |
Service de Neurochirurgie, Hôpital Pontchaillou, Rennes, France
Correspondence to:
Correspondence to:
S F A Amlashi
Service de Neurochirurgie, Hôpital Pontchaillou, Rue Henri Le Guilloux, 35033 Rennes cedex, France; amlashi@yahoo.com
Keywords: intracranial hypertension; giant
| The first 150 words of the full text of this article appear below. |
We read with great interest the article by Arjona et al1 concerning a patient with intracranial hypertension (ICH) and giant arachnoid granulations (GAG). The patient was a non-obese male without known risk factors for ICH apart from left transverse sinus hypoplasia and several GAGs in both transverse sinuses. The authors suggested that arachnoid granulations (AGs) might be responsible for ICH by obstructing the sinusal venous flow.
We would like to underline another point of view concerning such a physiological role for AGs. According to Clark,2 the AGs are absent at birth and develop in infants at the time of closure of the fontanelles. They increase in number and size with age and are commonly found in the vicinity of cortical venous entry sites into the sinuses, where there could be weaknesses in the dura mater. Thus they could be regarded as arachnoid herniations developing from intracranial CSF pulsation at higher
| HOME | HELP | FEEDBACK | SUBSCRIPTIONS | ARCHIVE | SEARCH | TABLE OF CONTENTS | REGISTER |