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NEUROLOGICAL PICTURE |
Department of Neurology, Graduate School of Medicine, Chiba University, Chiba, Japan, 1-8-1 Inohana, Chuo-ku, Chiba, 260-8670, Japan
Correspondence to:
Correspondence to:
Dr S Ito
Department of Neurology, Graduate School of Medicine, Chiba University, 1-8-1 Inohana, Chuo-ku, Chiba, 260-8670, Japan; sito@faculty.chiba-u.jp
Abbreviations: MRI, magnetic resonance imaging
Keywords: Longitudinal myelitis; Sjögrens syndrome
| The first 150 words of the full text of this article appear below. |
A 31 year old woman presented with sudden onset of complete paraplegia, decreased sensation for all areas below T7 spinal levels, and urinary retention. Spinal magnetic resonance imaging (MRI) showed multiple and confluent hyperintensities within the entire spinal cord extending into lower part of the medulla oblongata in T2 weighted images (fig 1
). T1 weighted, gadolinium enhanced, images also showed multiple confluent contrast enhanced lesions within the entire spinal cord, consistent with longitudinal myelitis (fig 2
). Laboratory data and a lip biopsy confirmed the diagnosis of Sjögrens syndrome. As no sicca symptoms were seen in this case, acute longitudinal myelitis was considered to be the initial manifestation of Sjögrens syndrome. Acute disseminated encephalomyelopathy and Devics disease were deemed unlikely because of the absence of brain and optic nerve lesions. Laboratory data excluded sarcoidosis, a disease of collagen, several infections, and metastatic tumours. Although a few cases have been
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