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Journal of Neurology, Neurosurgery, and Psychiatry 2002;73:349-350; doi:10.1136/jnnp.73.3.349
Copyright © 2002 by the BMJ Publishing Group Ltd.
Journal of Neurology Neurosurgery and Psychiatry 2002;73:349-350
© 2002 Journal of Neurology Neurosurgery and Psychiatry

LETTER

Hypoglycaemia induced by phenytoin treatment for partial status epilepticus

G Di Gennaro, P P Quarato, G B Colazza, A Mascia, F Mari and M Manfredi

IRCCS "NEUROMED", Pozzilli (IS), Italy and Department of Neurological Sciences, University of Rome "La Sapienza", Rome, Italy

Correspondence to:
Correspondence to:
Dr G Di Gennaro, IRCCS Neuromed, via Atinense, no 18, 86077, Pozzilli (IS), Italy;
gdigennaro@neuromed.it

Keywords: epilepsy; phenytoin; status epilepticus

The first 150 words of the full text of this article appear below.

A 22 year old woman was admitted at our epilepsy unit in status epilepticus. On examination, seizures were characterised by a confusional state with little response to external stimuli, and recurrent, brief, tonic motor manifestations lateralised to the left side. Family history was negative for epilepsy and metabolic disorders. Full term birth was uncomplicated and first psychomotor developmental milestones were normal. In the past medical history there was no sign of any metabolic diseases. There were no reports of cognitive dysfunction or personality disturbances. At the age of 16, the patient presented with epilepsy, which was characterised by two types of seizures: global tonic seizures, which occurred yearly, and brief episodes of loss of contact without any other manifestations, which were rare. The patient was treated for many years with 20 mg of clobazam twice daily. The awake EEGs that were performed routinely during the years of treatment with clobazam . . . [Full text of this article]


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