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Aggressive monitoring of electrocardiogram, blood pressure and blood glucose levels after 72 h in patients with insular infarcts is crucial in identifying whether they are prone to develop arrhythmias, hyperglycaemia and hypertension after the onset of stroke.
Infarction of the insular cortex may be associated with autonomic sympathetic activation, leading to repolarisation changes and arrhythmias.1 Moreover, the involvement of the insular cortex in the infarct area has been reported to be associated with a higher frequency of hyperglycaemia, and this association does not depend on the volume of the lesion, or pre-existing diabetes mellitus.2 Patients with insular infarcts also show a marked increase in nocturnal blood pressure, and higher serum …
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