Cinical PracticePredictors of chronic "postviral" fatigue
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Rehabilitation of Neuropsychiatric Symptoms in Patients With Long COVID: Position Statement
2023, Archives of Physical Medicine and RehabilitationBiopsychosocial risk factors of persistent fatigue after acute infection: A systematic review to inform interventions
2017, Journal of Psychosomatic ResearchCitation Excerpt :Petersen et al. [37] reported that pre-infectious fatigue was associated with sub-acute fatigue. In terms of chronic fatigue, pre-morbid fatigue was a risk factor in a higher quality, large community study [39] but not a smaller, primary-care study [31]. More general, pre-existing (chronic) health problems were associated with both chronic fatigue [36], and long-term fatigue [40].
Chronic Fatigue Syndrome
2017, Principles and Practice of Pediatric Infectious DiseasesChronic fatigue syndrome/myalgic encephalomyelitis (CFS/ME) is associated with pandemic influenza infection, but not with an adjuvanted pandemic influenza vaccine
2015, VaccineCitation Excerpt :There is still a lack of large, well-characterized cohort studies with CFS/ME as endpoint. Studies in general practice suggest that prolonged fatigue is not specifically related to features of common viral illnesses [36,37]. By following subjects with either acute Epstein–Barr virus infection, Q fever or Ross river virus infection in a rural region of Australia, it was found that 11% had prolonged illness with disabling fatigue [20].
Descriptive and predictive validity of somatic attributions in patients with somatoform disorders: A systematic review of quantitative research
2013, Journal of Psychosomatic ResearchChronic fatigue syndrome
2013, Handbook of Clinical NeurologyCitation Excerpt :More recently, prospective studies investigated whether acute viral infections could predict the onset of CFS. Two of the earlier studies showed that common viral infections, such as upper respiratory tract infections, were not associated with the subsequent development of either chronic fatigue or CFS, and concluded that viruses did not play a role in the onset of CFS (Cope et al., 1994; Wessely et al., 1995). Subsequent studies, however, showed that certain more severe infections played a role in the onset of CFS including infectious mononucleosis (glandular fever) (White et al., 2001; Moss-Morris and Spence, 2006), hepatitis (Berelowitz et al., 1995), viral meningitis (Hotopf et al., 1996), Q fever (Wildman et al., 2002), and Ross River virus (Hickie et al., 2006).