Table 2

Summary of electrophysiological data in patients with OPIDP

Case No (compound)Day after poisoningMotor2-150Sensory2-150Notes
NerveCV (m/s)CMAP (m/V)NerveCV (m/s)SAP (μV)
9 (Chlorpyrifos)24Common peroneal5015
Ulnar55-6020
62Common peroneal41-44 82-151
Ulnar48-52122-151 Ulnar442
10 (Methamidophos)10Common peroneal47 8Sural549
Ulnar534
34Common peroneal40 0.1Sural4411 §
Median60 3
Ulnar5415Ulnar556
11 (Isofenphos + phoxim)26Common peroneal22 0.3
Median50 0.3Median6013
700Common peronealNot measurable **
Median25 0.2Median7110
  • 2-150 Reference values were: motor conduction velocity (CV): common peroneal ⩾ 45, ulnar ⩾ 50, median ⩾ 48; sensory conduction velocity: sural ⩾ 48, ulnar ⩾ 53, median ⩾ 48; compound motor action potential (CMAP): common peroneal ⩾ 5, ulnar ⩾ 6, median ⩾ 7; sensory action potentials (SAP): sural ⩾ 6, ulnar ⩾ 4, median ⩾ 7.

  • 2-151 An increased frequency of polyphasic potentials was found. Fibrillation potentials were present in distal leg muscles, but not in arm muscles. § Fibrillation potentials were present in both leg and arm distal muscles. In subsequent electrophysiological studies motor and sensory CVs were not measurable and severe signs of denervation (fibrillation potentials) were found in arm, and especially, leg muscles. ** Signs of denervation (fibrillation potentials) were still present and much less evident in both arm (only distally) and leg muscles. Partial reinnervation took place (scarce interference pattern with high frequency potentials) in arm muscles and to a lesser extent in proximal leg muscles.