Conduction velocity and conduction block after experimental ischaemic nerve injury☆
References (17)
- et al.
Acute ischaemic neuropathy in the rabbit
J. neurol. Sci.
(1979) - et al.
Peripheral nerve ischemia, Part 1 (Experimental model)
J. neurol. Sci.
(1975) - et al.
Acute peripheral nerve compression in the baboon
J. neurol. Sci.
(1974) - et al.
Acute nerve compression during limb ischaemia
J. neurol. Sci.
(1980) The blood supply of nerves
J. Anat. (Lond.)
(1943)Experimental Acute Ischaemic Neuropathy in the Rabbit
- et al.
Recovery of nerve conduction after a pneumatic tourniquet — Observations on the hind-limb of the baboon
J. Neurol. Neurosurg. Psychiat.
(1972) Acute compression block
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Hearing outcomes after loss of brainstem auditory evoked potentials during microvascular decompression
2015, Journal of Clinical NeuroscienceCitation Excerpt :In acute conditions where pressure of high magnitude has been directly applied to a nerve trunk, the resulting disturbances in nerve function are based mainly upon the effect of myelin damage. The redistribution of tissue from compressed to non-compressed levels of the nerve is associated with invagination of the myelin sheaths at the nodes of Ranvier [21,22]. The resulting myelin damage is associated with partial or complete local conduction block and it is usually reversible.
Neurophysiologic responses of peripheral nerve to repeated episodes of anoxia
2013, Clinical NeurophysiologyCitation Excerpt :The nervous system is especially vulnerable to hypoxia and ischemia (Astrup et al., 1977; Harden et al., 1966). There have been a large number of in vivo studies of the response of the peripheral nervous system in humans to ischemia using the tourniquet-ischemia model (CruzMartinez et al., 1980; Caruso et al., 1973; Abramson et al., 1971; Seneviratne and Peiris, 1968; Schmelzer et al., 1989; Stohr, 1981; Korthals and Wisniewski, 1975; Bostock et al., 1994) and a growing number of studies on ischemia using in vivo animal models (Schmelzer et al., 1989; Fowler and Gilliatt, 1981; Low et al., 1985, 1986; Kihara et al., 1996; Mitsui et al., 1999). These studies have demonstrated the initial electrophysiologic changes of ischemia appear roughly 10–30 min after onset and include decline in the NAP amplitude and velocity.
Pseudo-conduction blocks in a case with polyarteritis nodosa
2012, Neurology Psychiatry and Brain ResearchCitation Excerpt :Both reversible and persistent conduction blocks may occur in animal models with peripheral nerve ischemia without complete cutting of the axon. There have been relatively few studies on conduction blocks in human vasculitic neuropathy.1–3 Here, we present a case indicating distal Wallerian degeneration following focal axonal conduction disturbances.
Recombinant interferon-beta therapy and neuromuscular disorders
2009, Journal of NeuroimmunologyInactivation of baroafferents leads to loss of barosensitivity without changes in nerve morphology
1998, Journal of the Autonomic Nervous SystemPhotochemically induced experimental ischemic neuropathy: A clinical, electrophysiological and immunohistochemical study
1993, Journal of the Neurological Sciences
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This work was supported financially by the Medical Research Council of Great Britain.