Conduction block without demyelination following acute nerve infarction
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Cited by (42)
The electrophysiology of axonal neuropathies: More than just evidence of axonal loss
2020, Clinical NeurophysiologyCitation Excerpt :Interestingly, the nerve fibres of older subjects were more resistant to the anoxic-ischaemic stress (Caruso et al., 1973), and it has been suggested that the increasing resistance to ischemia with aging is related to a progressive reduction of energy requirements (Low et al., 1986). Transient or long lasting CB has been also reported following experimental acute occlusion of vasa nervorum by injecting arachidonic acid (Parry and Linn, 1988). Morphological examination did not reveal segmental demyelination suggesting a “metabolic” nature for CB.
Conduction block: A notion to let through
2012, Revue NeurologiqueNerve conduction studies: Basic concepts and patterns of abnormalities
2012, Neurologic ClinicsCitation Excerpt :Conduction block represents a focal pathologic process affecting peripheral nerve. However, although conduction block is generally used interchangeably with suspected underlying segmental demyelination, axonal pathology may also create clinically and electrophysiologically observable conduction block.48,49 For example, acute focal axonal lesions and chronic immune-mediated neuropathies such as multifocal motor neuropathy with conduction block may both be characterized by some degree of axonally mediated conduction block.35,47
Neuropathies associated with medical conditions
2006, Handbook of Clinical NeurophysiologyNerve conduction studies: Types, components, abnormalities, and value in localization
2002, Neurologic ClinicsSensitivity and specificity of different conduction block criteria
2000, Clinical NeurophysiologyCitation Excerpt :In keeping with the fact that conduction blocks are focal in MMNP and disseminated in AIDP, ratios for the whole arm amplitude decay (Erb's point – wrist) were more sensitive in AIDP, whereas focus on the upper arm segment increased sensitivity in MMNP. We use criterion D in chronic neuromuscular diseases, when motor unit restructuring and delayed conduction have to be taken into account, and we use the less strict limits derived from healthy controls (criterion A) in acute conditions like AIDP or acute nerve infarction (Parry and Linn, 1988). Measurement difficulties alone explain why conduction block criteria have to be stricter in small responses.