Research articleCorticonigral degeneration with neuronal achromasia presenting with primary progressive aphasia: Ultrastructural and immunocytochemical studies☆
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2008, Handbook of Clinical NeurologyCitation Excerpt :In most cases, however, neuronal loss is not apparent (Kikuchi et al., 1999; Iwasaki et al., 2005). The expanded cytoplasm of the cortical ballooned cells is very weakly argyrophilic and stains positively and strongly for phosphorylated neurofilaments (Dickson et al., 1986; Smith et al., 1992; Arima et al., 1994) that do not extend into the swollen proximal processes of the ballooned neurons. They are also immunoreactive for alpha β‐crystalline (Kato et al., 1992; Lowe et al., 1992), heat shock protein 27 (Kato et al., 1992) and variably with antibodies to ubiquitin on epitopes different from AD's NFTs (Dickson et al., 1986; Feany and Dickson, 1995; Halliday et al., 1995).
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This study was presented in abstract form at the 12th International Congress of Neuropathology, September 20, 1994 (Brain Pathology, 4 (1994) 515).