Original articleCytogenetic analysis of 39 pediatric central nervous system tumors
References (32)
Genes, chromosomes and cancer
J Pediatr
(1984)Role of chromosome translocation in human neoplasia
Cell
(1987)- et al.
The tumor phenotype and the human gene map
Cancer Genet Cytogenet
(1983) - et al.
Progress in the management of childhood brain tumors
Hematol Oncol Clin North Am
(1987) Role of chromosome translocations in human neoplasia
Cell
(1987)- et al.
Translocation involving chromosome 22 in Ewing's sarcoma. A cytogenetic study of four fresh tumors
Cancer Genet Cytogenet
(1984) - et al.
Chromosome study of Ewing's sarcoma (ES) cell lines. Consistency of a reciprocal translocation t(11;22)(q24;q12)
Cancer Genet Cytogenet
(1984) The involvement of oncogenes and suppressor genes in human neoplasia
Adv Pediatr
(1987)- et al.
Cytogenetic studies in twenty human brain tumors: Association of no. 22 chromosome abnormalities with tumors of the brain
Cancer Genet Cytogen
(1980) - et al.
Cytogenetic studies of human brain tumors and their clinical significance. II. Meningioma
Cancer Genet Cytogenet
(1987)
Establishment of a rhabdoid tumor cell line with a specific chromosome abnormality, 46,XY,t(11:22)(p15.5;q11.23)
Cancer Genet Cytogenet
Chromosomes and cancer: New nomenclature and future directions
Hum Pathol
Biological implications of consistent chromosome rearrangements in leukemia and lymphoma
Cancer Res
A cellular oncogene is translocated to the Philadelphia chromosome in chronic myelocytic leukemia
Nature
Human c-myc onc gene is located in the region of chromosome 8 that is translocated in Burkitt lymphoma cells
Oncogene activation by chromosome translocation in human malignancy
Annu Rev Genet
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Cathepsin L silencing increases As<inf>2</inf>O<inf>3</inf> toxicity in malignantly transformed pilocytic astrocytoma MPA58 cells by activating caspases 3/7
2017, Experimental Cell ResearchCitation Excerpt :Radiation induces the formation of dicentric chromosomes [38,39], which were present in the karyotype of the MPA58 cells. The MPA58 karyotype also included deletions, which are characteristic of PAs [40,41] and anaplastic astrocytomas [42]. As the aim of this study was to evaluate the sensitivity of primary malignant glioma cells to As2O3 toxicity, and to determine whether their chemosensitivity is modulated by CatL silencing, CatL expression was first assessed in several GBMs and the MPA58 sample.
Pediatric low-grade gliomas: How modern biology reshapes the clinical field
2014, Biochimica et Biophysica Acta - Reviews on CancerCitation Excerpt :The most frequent recurrent chromosomal alteration identified was a gain of chromosome 7, especially in PAs [174,177–179]. Other chromosomal structural abnormalities included gains of chromosomes 4, 5, 6, 8, and 11 and deletion of 17p in a subset of PAs, inversion in chromosome 8, and loss of chromosome 1q [174,177–185]. Genetic alterations in pediatric LGGs differ from adult LGGs.
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2012, Brain TumorsCraniopharyngiomas
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