You are here

Article Text

Article menu
Download PDFPDF
Letters to the editor
Isolated infarction in the territory of lateral posterior choroidal arteries
  1. NAOKATSU SAEKI,
  2. KATSUNORI SHIMAZAKI,
  3. AKIRA YAMAURA
  1. Department of Neurological Surgery Chiba University School of Medicine, 1–8–1 Inohana, Chuoh-ku, Chiba-shi, Chiba Japan 260–8670
  1. Naokatsu Saeki, MD, Department of Neurological Surgery, Chiba University School of Medicine, 1–8–1 Inohana, Chuoh-ku, Chiba-shi, Chiba, Japan 260–8670. Telephone 0081 43 226 2158; fax 0081 43 226 2159.

https://doi.org/10.1136/jnnp.67.3.413

Statistics from Altmetric.com

    Request Permissions

    If you wish to reuse any or all of this article please use the link below which will take you to the Copyright Clearance Center’s RightsLink service. You will be able to get a quick price and instant permission to reuse the content in many different ways.

    Lateral posterior choroid arteries (LPCAs) originate from the distal PCA trunk in the proximity of the origin of thalamogeniculate arteries or the cortical branches.1 Infarction of LPCAs, therefore, often accompany lesions of other PCA cortical branches or perforating branches.2 3 Their clinical features have often been masked by the associated and combined cortical and subcortical lesions. Accordingly the clinical features of a discrete infarction of the LPCA have been reported only a few times.2-5 This paper reports on two such patients and pertinent articles are reviewed.

    (A, B, C) Patient 1. CT taken 5 days after the onset. The low density lesion was seen at the posterior part of the right caudate nucleus and dorsolateral thalamus. (D) patient 1. MRI taken a month later. T1 weighted coronal image showed that the lesion was located at the posterior part of the caudate nucleus(arrow), the thalamus (arrows), the lateral geniculate body, and the part of medial temporal lobe (arrow head). (E, F) patient 2. CT taken 3 days after the onset. Low density lesions were seen at the posterolateral thalamus (arrow) and the posterior part of the caudate nucleus (arrows).  

    The first patient, a 62 year old woman with a history of untreated supraventricular arrhythmia for 10 years had sudden onset of non-pulsatile headache on the vertex. At the same time she noted blurred vision in the …

    View Full Text