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A 40-year-old, right-handed man was admitted to the EEG monitoring unit for localisation of epileptic focus and changes in medication regimen. The patient had undergone a left frontal-lobe resection during childhood for unknown pathology (figure1A). Subsequently he developed secondary epilepsy characterised by nocturnal complex partial seizures with occasional secondary generalisation. During the first 2 days of monitoring, a persistent myogenic artefact was seen localised to the T3 EEG electrode (figure 1C). The artefact prevented localisation of the patient's clinical seizure during monitoring. We attributed it to excessive muscle activity, as electrode lead replacement and …
Competing interests None.
Patient consent Obtained.
Provenance and peer review Not commissioned; externally peer reviewed.