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Editorial commentary
Post-stroke fatigue: new evidence of a possible biological cause
  1. Gillian E Mead
  1. Correspondence to Dr Gillian E Mead, Department of Geriatric Medicine, University of Edinburgh, Room S1642, Royal Infirmary, Little France Crescent, Edinburgh EH16 4SA, UK; gillian.e.mead{at}ed.ac.uk

https://doi.org/10.1136/jnnp-2015-310909

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    Impaired corticomotor excitability might contribute to the aetiology of post-stroke fatigue, and provide a biological target for treatment.

    Fatigue after stroke affects around 40% of stroke survivors and is one of the most distressing post-stroke symptoms.1 It may resolve spontaneously over time, but for many patients, it remains a persistent and disabling problem. There are no effective treatments. Stroke survivors, caregivers and health professionals have rated fatigue as one of the top 10 priorities for stroke research.2 Some stroke survivors report that fatigue starts at the time of the stroke, and that the nature of fatigue is unlike any fatigue experienced prior to their stroke. This would be consistent with the …

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    Footnotes

    • Competing interests I have been invited to mentor Dr Kuppuswamy for future grant applications, but I have had no input whatsoever into the current work on which this editorial is based.

    • Provenance and peer review Commissioned; internally peer reviewed.

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