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Emerging phenotype of MOG antibody-associated disease: acute haemorrhagic leucoencephalitis
  1. Sohyeon Kim,
  2. Sung Jo Bang,
  3. Jae-Joon Lee,
  4. Hung Youl Seok
  1. Department of Neurology, Keimyung University School of Medicine, Daegu, Korea (the Republic of)
  1. Correspondence to Professor Hung Youl Seok, Department of Neurology, Keimyung University School of Medicine, Daegu 42601, Korea (the Republic of); shy2354{at}

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Myelin oligodendrocyte glycoprotein (MOG) antibody-associated disease (MOGAD) has been extensively studied, revealing its expression in a variety of phenotypes.1 These include optic neuritis, myelitis, acute disseminated encephalomyelitis and other manifestations beyond those observed in neuromyelitis optica spectrum disorders.1 These broader clinical features have been well documented in the literature. More recently, additional manifestations of MOGAD have emerged, including cortical encephalitis phenotypes such as unilateral cortical fluid-attenuated inversion recovery-hyperintense lesions in anti-MOG-associated encephalitis with seizures.1

In a notable contribution to the growing body of knowledge, Skarsta et al introduced acute haemorrhagic leucoencephalitis (AHLE) as a novel phenotype associated with MOGAD in their publication in JNNP.2 Our recent findings have …

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  • Contributors HYS conceived and designed the research. All authors reviewed the original article and drafted the manuscript.

  • Funding The authors have not declared a specific grant for this research from any funding agency in the public, commercial or not-for-profit sectors.

  • Competing interests None declared.

  • Provenance and peer review Not commissioned; internally peer reviewed.