Table 1

Clinical features, electrophysiological patterns at final diagnosis and laboratory findings

Patient NoParaesthesiaPainSensory lossDTRNeurophysiological classificationSignificant SNAPa changesAntecedent infectious symptomsAntiganglioside IgG
GM1GD1aGD1b
1AMAN/ADDiarrhoea/fever
2*AMAN/ADDiarrhoea51 200800
3+AMAN/AD+Diarrhoea/feverNDNDND
4AMAN/AD+Diarrhoea12 800
5+AMAN/AD+Flu16006400
6+AMAN/AD+Diarrhoea/fever1600
7++AMAN/AD+Diarrhoea/fever160051 200
8*AMAN/AD+Diarrhoea320012 800
9+++AMAN/RCF+Diarrhoea51 200102 400102 400
10+AMAN/RCFDiarrhoea51 200102 400
11++AMAN/RCFDiarrhoea/fever400
12AMAN/AD/MSA+Diarrhoea
13+AMAN/AD/MSA+Diarrhoea64001600400
14++AMSAN/ADURTI400400800
15*++AMSAN/AD+Diarrhoea51 20012 800
16*+++AMSAN/RCF+Diarrhoea/fever400400
  • The titre of IgG to gangliosides is reported. Paraesthesia and pain were reported to be present (+) or absent (−). DTR were absent/hypoactive (−), preserved (+) or brisk (++).

  • * Patients who were prospectively evaluated.

  • AD, axonal degeneration pattern in motor fibres; AMAN, acute motor axonal neuropathy; AMSAN, acute motor and sensory axonal neuropathy; DTR, deep tendon reflexes; MSA, minor sensory abnormalities at baseline; RCF, reversible conduction failure pattern in motor fibres; SNAPa, sensory nerve action potential amplitude; URTI, upper respiratory tract infection.