Case reportDiphtheritic myocarditis: A histochemical and electron microscopic study☆
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Cited by (20)
DIPHTHERIA
2009, Feigin and Cherry's Textbook of Pediatric Infectious Diseases, Sixth EditionDiphtheria toxin-induced autophagic cardiomyocyte death plays a pathogenic role in mouse model of heart failure
2004, Journal of Biological ChemistryCitation Excerpt :Myocardial involvement is a major complication that determines the prognosis, but the pathophysiology associated with diphtheritic cardiomyopathy remains largely unknown. Histological analysis of the post-mortal heart of a diphtheria patient revealed hyaline degeneration of myocardium and infiltration of mononuclear cells, and cytosolic lipid droplets and clumped chromatin granules were observed by electron microscopic examination (51). Although vacuoles of autophagosomes were not described in this paper, these findings were similar to those observed in our mouse model.
The effects of diphtheria toxin on guinea-pig myocardial protein synthesis
1973, Journal of Molecular and Cellular CardiologyCardiomyopathies in infants and children
1972, Progress in Cardiovascular DiseasesDiphtherial myocardiopathy
1972, The American Journal of CardiologySpecific reversal of diphtheria toxin mediated inhibition of protein synthesis in guinea pig tissues
1970, Biochemical and Biophysical Research Communications
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This study was supported by grants from the U. S. Public Health Service, the Rudolph Matas Memorial Fund for the Kate Prewitt Hess Laboratory and the Rowell A. Billups Fund for Research in Heart Disease.