Ischaemic cerebrovascular diseases in an autopsy series Part 2. Prevalence, location, pathogenesis, and clinical course of cerebral infarcts
References (104)
- et al.
Observations on the natural history of transient cerebral ischaemia
Lancet
(1964) - et al.
Factors influencing the prognosis of cerebral thrombosis and infarction due to atherosclerosis
J. chron. Dis.
(1960) L'ictus apoplectique, problème de gérontologie
Schweiz. med. Wschr.
(1959)Course and prognosis in spontaneous occlusions of cerebral arteries
Acta psychiat. neurol. scand.
(1958)The natural history of transient ischaemic cerebrovascular attacks
Quart. J. Med.
(1964)- et al.
Patterns of vascular failure in the central nervous system
J. Neuropath. exp. Neurol.
(1954) Circulatory changes following occlusion of the middle cerebral artery and their relation to function
J. Neurosurg.
(1958)- et al.
Cerebral infarction by atheromatous emboli
Report of case and review of literature
Arch. Path.
(1963) - et al.
Cerebral embolism in cardiac disease — the clinical and pathological studies of 56 cases
Jap. Heart J.
(1960) - et al.
Comparative incidence of cerebrovascular disease in a general hospital in Boston and in Oslo
Ischemic cerebral lesions
Angiology
The effects of systemic hypotension upon the human brain
Clinical and neuropathological observations in 11 cases
Brain
Vascular diseases of the brain
Bull. New Engl. med. Cent.
Vascular diseases of the brain
Bull. New Engl. med. Cent.
Vascular diseases of the brain
Bull. New Engl. med. Cent.
Pathology of cerebral arterial occlusion
Progressing stroke: Pathogenesis
Vascular diseases of the brain
Ann. Rev. Med.
Little Strokes
Stenosis and occlusion of vessels in cerebral infarction
Brit. med. J.
Arteriographic study of cerebrovascular disease, Part 2 (Cerebral symptoms due to kinking, tortuosity, and compression of carotid and vertebral arteries in the neck)
Arch. Neurol. (Chic.)
The blood pressure in a population
Blood pressure readings and height and weight in the adult population of the city of Bergen
Acta med. scand.
The agonal nature of cerebral red softenings: Re-evaluation of the morphological findings
Acta psychiat. neurol. scand.
Ingravescent cerebral infarction
Quart. J. Med.
Cerebral hemorrhage from venous and capillary stasis
Amer. J. med. Sci.
Cerebrovascular accidents
A study of two hundred and one cases
Arch. Path.
Comparative incidence of cerebrovascular lesions in normotensive and hypertensive patients
Neurology (Minneap.)
The clinical aspects of cerebral vascular insufficiency
Ann. intern. Med.
Pathology of the Central Nervous System
Cerebral embolism
Angiographic observations on spontaneous clot lysis
Lancet
Survey of 1000 cases of apoplexia cerebri
Acta psychiat. neurol. scand.
Fatal atheromatous cerebral embolism associated with bright plaques in the retinal arterioles
Report of a case
Neurology (Minneap.)
Recurrent cerebrovascular episodes
Arch. Neurol. (Chic.)
The cerebral collateral circulation, Part 2 (Production of cerebral infarction by ischemic anoxia and its reversibility in early stages)
Neurology (Minneap.)
Carotid kinking as a cause of cerebral insufficiency
Circulation
Embolization and transient blindness from carotid atheroma
Surgical considerations
Arch. Surg.
Apoplexy
Pathogenesis
Acta psychiat. neurol. scand.
Histologic studies of the brain following head trauma, Part 3 (Post-traumatic infarction of cerebral arteries, with consideration of the associated clinical picture)
Arch. Neurol. Psychiat. (Chic.)
Pathogenesis of hemorrhagic infarction of the brain. Part 1 (Experimental investigations of role of hypertension and of collateral circulation)
Arch. Neurol. (Chic.)
Radiologic visualization of neck vessels in healthy men
Neurology (Minneap.)
Experimentally produced red softening of the brain
J. Neuropath. exp. Neurol.
Occlusion of the carotid arteries
Further experiences
Arch. Neurol. Psychiat. (Chic.)
Observations of the fundus oculi in transient monocular blindness
Neurology (Minneap.)
Clinical syndromes in cerebral arterial occlusion
Lacunes: Small, deep cerebral infarcts
Neurology (Minneap.)
Observations on brain embolism with special reference to the mechanism of hemorrhagic infarction
J. Neuropath. exp. Neurol.
Atherosclerosis of the carotid and vertebral arteries — extracranial and intracranial
J. Neuropath. exp. Neurol.
The nonsudden onset of cerebral embolism
Neurology (Minneap.)
Microscopical observations on the circulation of the blood in the cerebral cortex
Brain
Contribution à l'étude du ramollissement cérébral envisagé au point de vue de sa fréquence, de son siège et de l'état anatomique des artères du territoire nécrosé
J. Neurol. (Brux.)
Les infarcissements hémorragiques de l'écorce cérébrale
Étude anatomoclinique et statistique
Acta neuroveg. (Wien)
Spontane cerebellumblødninger og emollisjoner
Nord. Med.
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