HypothesisALCOHOLIC DEMENTIA: A HYPOTHESIS
References (31)
- et al.
The investigation of dementia-results in 200 consecutive admissions
Lancet
(1981) - et al.
Brain lesions in alcoholics. A neuropatholgical study with clinical correlations
J Neurol Sci
(1982) Magnocellular nuclei of the basal forebrain project to neocortex, brain stem, and olfactory bulb Reviw of some functional correlates
Brain Res
(1975)- et al.
A cytoarchitectonic and histochemical study of nucleus basalis and associated cell groups in the normal human brain
Neuroscience
(1984) Clinical spectrum and epidemiological features of alcoholic dementia
The relationship between Korsakov's syndrome and 'alcoholic dementia'
Br J Psychiatry
(1978)- et al.
The alcoholic dementias
Brain damage in chronic alcoholics: a review ofthe psychological evidence
Objective psychological changes in alcoholics after the withdrawal of alcohol
Br Med Bull
(1982)Cognitive impairment in chronic alcoholics. Some cause for optimism
Am Psychol
(1983)
Cognitive loss and recovery in long-term alcohol abusers
Arch Gen Psychiatry
Computerised tomography of the brain in chronic alcoholism A survey and follow-up study
Brain
The alcoholic brain: CT scan and psychological findings
Psychol Med
Brain weights in alcoholics (short report)
J Neurol Neurosurg Psychiatry
Brain atrophy in chronic alcoholic patients—a quantitative pathological study
J Neurol Neurosurg Psychiatry
Cited by (63)
Alcohol-related amnesia and dementia: Animal models have revealed the contributions of different etiological factors on neuropathology, neurochemical dysfunction and cognitive impairment
2011, Neurobiology of Learning and MemoryCitation Excerpt :This quandary results in problems for the clinical diagnosis of both syndromes (Harper, 1998; Olsin et al., 1998. Others (Lishman, 1986; Lishman, 1990; Smith & Atkinson, 1995) argue that AAD and WKS are distinct disorders with overlapping clinical symptoms. This “dual vulnerability hypothesis” states that the development of these disorders in certain individuals likely involves numerous factors.
Alcohol and the Nervous System
2008, Neurology and General MedicineToxic dementias
2008, Handbook of Clinical NeurologyCitation Excerpt :In this chapter we will first review the dementia of alcohol abuse and then consider the effects of exposure to heavy metals, industrial solvents, carbon monoxide poisoning, misuse of prescription medications, and illicit drug abuse on cognition and the development of dementia. Individuals who consume large amounts of alcohol have an increased risk of cognitive impairment (Lishman, 1981; Butters, 1985; Goldstein, 1985; Lishman, 1986; Ryan and Butters, 1986; Nutt, 1999; Oslin, 2003). The best‐documented alcohol disorder is Wernicke–Korsakoff syndrome (WKS), which typically strikes individuals in mid‐life, after two or three decades of heavy drinking, and is associated with thiamine deficiency.
Alcohol and the Nervous System
2007, Neurology and General Medicine: Expert Consult